RATDP Overview

The Regional Adipose Tissue Deposit Profile

There is always a great debate in health sciences regarding the ability to define a best practice for each individual. The same questions are constantly being asked…

  • Can you can do something specific to take weight off that is superior to other options?
  • Can you take weight off of a specific spot on the body?
  • Can you perform a type of workout that is best for you?

The traditional concept of exercising a body part harder and longer to take fat off of a specific area of the body has shown limited support in the scientific literature. Attempting to use more fat substrates in the intramuscular tissue of a specific muscle group when exercising a specific area is a poor strategy for improvement but the macro theory of looking at how and where fat deposits on the body has much more scientific validity. 

"It is becoming increasingly evident that cardiovascular risk is not simply linked to the quantity of adipose tissue, but more importantly the location in which it accumulates. Regional fat distribution appears to play a significant role in cardiovascular disease and recent work has shown that regional fat deposits have a variety of functions other than simply a storage depot for fat."

Epicardial adipose tissue: far more than a fat depot

Cardiovasc Diagn Ther. 2014 Dec; 4(6): 416–429.

doi:  10.3978/j.issn.2223-3652.2014.11.05


Unlike other methods that try and ascertain specific hormonal imbalances, the RATDP methodology steps back and looks at the overall interpretation that the shape of the body is telling us in terms of the known scientific principles that cause that type of human morphological profile. Six Sigma Fitness™ doesn't focus on just the cause but also the effect.  In other words if "A" causes "B" and root cause analysis proves this out then eliminating or changing "A" is instrumental to the long-term solution.  But that being said, we also need to look at what "B" is causing and treat that as well to get results.  So if A causes B and B causes C we need to address both A and C to solve the problem as it applies to human chemistry.

Scientific Explanation:  "...individuals with visceral abdominal obesity have been shown to be more insulin resistant than those with peripheral obesity, have lower glucose disposal during an euglycemic (normal blood sugar) hyperinsulinemic (high insulin) clamp, reduced oxidative and nonoxidative glucose and leucine disposal, and significantly greater lipid oxidation. These results suggest that an excess of visceral fat results in a detrimental effect on glucose and protein metabolism, with the excess of Free Fatty Acids being the link between central fat and insulin resistance."
Subcutaneous and Visceral Adipose Tissue: Their Relation to the Metabolic Syndrome

Layman's Terms: We used to believe that if  you demonstrated accumulation of abdominal fat that this was a cortisol issue that could be addressed separately and in isolation from all other aspects of human chemistry. We now know that cortisol imbalance can cause a host of other problems including insulin resistance which is a form of metabolic syndrome and the early path to Type 2 diabetes which also causes abdominal and "muffin top" fat to accumulate.  Therefore we need to also now address the insulin resistance and the reduced ability to use glucose (sugars) for energy by potentially reducing the quantity and increasing the quality of carbohydrates in the diet while performing whole-body resistance training.  More endurance training may only stress the body further and slow endurance may have little effect on insulin receptors.  In addition, certain lifestyle habits such as a lack of sleep may be addressed as it stresses the body as well and has shown to create insulin resistance. The recent science suggests that once you develop abdominal obesity it is not only about cortisol but also about becoming insulin resistant and unable to adequately use glucose for energy. Multiple problems creep up that are making the original issue difficult to address in isolation.

In the scientific example explained above, the root cause of the visceral fat must be addressed which in the above case may be stress induced high cortisol, but the effect that the visceral fat has caused on insulin receptors must now also be addressed to help stimulate results. The effects of the insulin resistance may be the most pronounced issue that must be addressed but as it is normalized the abdominal fat causes will quickly become the next priority and as such would most likely respond best when treated together.

The rank prioritization and accompanying analytical reports in the Six Sigma Fitness™ technology will make this determination and focus the protocols on those issues that may be of greatest opportunity to achieve results.  Suffice to say the answer is never one chemical or hormone creating one issue but a cascade of effects with each one becoming the root cause of the next effect.  The key is not to be ignorant of what may be happening chemically and not recommended protocols or programming that may slow or impede progress.  Sometimes it's as much about what you don't do than what you do do.

Due to the complicated science of hormonal physiology, science is still trying to understand and discover all of its affects to the body. This specific concept of regional adipose tissue deposits is still in its infancy but being researched extensively. As such there are many macro clues and scientific theories to look to that would answer why the concepts of how regional adipose (fat) tissue deposition can help determine an optimal course of action for each individual. More on that body of scientific research in the final lesson of this mini introductory course.